In a prostate cancer-prone Pten-/- mouse model, FA translocase has been shown to facilitate FA uptake and storage, significantly impacting fatty acid oxidative (FAO) metabolism and reversing increases in acylcarnitines, monoacylglycerols, and phospholipid hydrolysates induced by Pten deficiency[90]. The gene discussed is PTEN; the disease is Familial prostate cancer.