(128) found that treating Ldlr-/-rag1-/- mice with IL-2/anti-IL-2 complexes resulted in the expansion of CD25+ ILC2s, which lowered very low-density lipoprotein cholesterol and atherosclerosis, and conversely ILC2s depletion led to an acceleration of the atherosclerotic process (129). Here, IL2 is linked to atherosclerosis.