We previously reported that NSA2, a putative cytosolic ribosomal biogenesis factor (Glasgow et al. 2017), which had been shown to be involved in cell cycle regulation and suggested as a putative protooncogene, was expressed at higher levels in diabetic mouse kidneys, in renal cells exposed to diabetic conditions, and in blood samples from patients with diabetic kidney disease (Shahni et al. 2012, 2013; Wu et al. 1999; Zhang et al. 2010). Here, NSA2 is linked to diabetic kidney disease.