In contrast, phospho‐p38MAPK, a marker of non‐canonical TGFβR1/ALK5 receptor signaling, was unchanged in BLEO‐IPF mice and not influenced by ALK5i (Figure S8b), suggesting preferential recruitment of canonical TGFβR1/ALK5 receptor signaling upon ALK5i treatment. The gene discussed is TGFBR1; the disease is idiopathic pulmonary fibrosis.