Most of them corresponded to proteinsthat increased with the severity of the infection (UBR4, XIAP, NDUAA,RBM4B, RHG35, SCYL1, FKB15, AN13A, EPS8, NXP20, CC186, and CPSF7; Figure 2a), and exclusivelyone protein (KIF3B) displayed opposite behavior (Figure 2b). The gene discussed is XIAP; the disease is infection.