MCL1 and infection: Genes upregulated for the BCL-2 family of antiapoptotic proteins (MCL1 in LMH cells, and BCL2A and BCL2L14 in CEK cells infected with either strain of ILTV) [84,85], were in agreement with the notion that ILTV infection prevents apoptosis of infected cells, in order to prolong viral replication [11].