The prominence of interferons (IFNs) in the mouse model [including IFNG and the Interferon alpha and Interferon alpha and beta receptor (Ifnar) groups] mirrors features of human endometriosis, where elevated IFNA1/2 and IFNAR2 mRNA are reported in the eutopic endometrium compared to in the endometrium of women without disease (Kao et al., 2003). The gene discussed is IFNAR2; the disease is endometriosis.