Lgals1 was also highly expressed in monocytes, pro-monocytes and GMPs, but neither the abundance of these cell types nor the per cell Lgals1 expression was increased in myelofibrosis, indicating that monocytes and their precursors are not a source of excess galectin-1 production in myelofibrosis (Fig. S5F). The gene discussed is LGALS1; the disease is myelofibrosis.