Patients with myositis share some common clinical features, including muscle weakness, increased circulating muscle enzymes (e.g., creatine kinase), inflammatory infiltration of CD4+/CD8+ T‐cells and B‐cells within the muscle, expression of muscle‐specific and ‐associated autoantibodies and interferon (IFN) upregulation in muscle fibers, and overexpression of major histocompatibility complex class I (MHC‐I) on the surface of muscle fibers (Carstens & Schmidt, 2014). This evidence concerns the gene IFNA1 and myositis disease.