GAD1 and insomnia: Through HE staining, we found that the pathological changes in the CA1 region of the hippocampus were particularly obvious, and GAD67 was significantly decreased in the insomnia group, overexpression of GAD67 would increase its expression, and senegenin was further up‐regulated after intervention, which indicated that senegenin could improve insomnia by regulating GAD67, and was closely related to hippocampal tissue.