WTAP and metabolic dysfunction-associated steatohepatitis: CDK9‐mediated phosphorylation of WTAP drives its translocation from the nucleus to the cytosol, a pivotal modification that significantly influences WTAP's regulatory functions in NASH.[92] In addition, IFN‐γ activates ERK signaling, leading to WTAP phosphorylation, which enhances m6A modification of immunosuppressive molecules like IDO1, PD‐L1, ICAM1, and VCAM1 in mesenchymal stem cells (MSCs).