This modification boosts the stability of MCM2 and MCM5 mRNAs, facilitating cell cycle progression and tumorigenesis in HBV‐related HCC.[158] Moreover, at the onset of type 1 diabetes (T1D), METTL3 in pancreatic β‐cells is upregulated, enhancing m6A methylation of key immune mediators and controlling the antiviral response. This evidence concerns the gene MCM2 and type 1 diabetes mellitus.