This modification boosts the stability of MCM2 and MCM5 mRNAs, facilitating cell cycle progression and tumorigenesis in HBV‐related HCC.[158] Moreover, at the onset of type 1 diabetes (T1D), METTL3 in pancreatic β‐cells is upregulated, enhancing m6A methylation of key immune mediators and controlling the antiviral response. The gene discussed is MCM5; the disease is type 1 diabetes mellitus.