Indeed, this comparative study shows that, in our cellular models of prostate cancer, there is no significant difference in terms of functionality, regulatory pathways or interface with MET between ETV1 and ERG, which would suggest a common role in prostate cancer, the specificity being rather linked to the fact that the tumours present one or other of the gene fusions, knowing that their presence is mutually exclusive [67]. The gene discussed is ERG; the disease is neoplasm.