The predominant predicted tumor-to-microglia interactions in this model were driven by tumor junctional adhesion molecule (JAM) and extracellular matrix (ECM) ligands, such as collagens (e.g., COL4A2, COL6A3) and laminins (e.g., LAMA3, LAMC1; Figure 6C). The gene discussed is F11R; the disease is neoplasm.