For instance, the engineering of macrophages with the cytosolic domain from MERTK, which has been described to trigger tumour cell cytotoxicity via targeting of immunosuppressive CCR7+ cells inside the tumour,10 might exert its effect in prolonging survival in mouse models also because of the post‐engulfment consequences of taking up apoptotic cells with specific identities. Here, CCR7 is linked to neoplasm.