Here, strong associations were observed with sun exposure, high CDKN1A expression, and SenSkinTM, which included SASP genes, suggesting that sun exposure, like other damage stimuli such as fracture (Chandra et al., 2022; Saul et al., 2024) or acute lung injury (Blázquez‐Prieto et al., 2021), tended to activate p21‐related pathways of cellular senescence. The gene discussed is CDKN1A; the disease is injury.