BTG3 and lymphangioleiomyomatosis: They suggested two variants: an inflammatory and a non-inflammatory.3 Similarly, Utikal et al. reported patients with marked telangiectatic erythema within lesions of linear atrophoderma, suggesting a novel variant.4 The exact etiology of LAM remains unknown but is postulated to result from somatic mosaicism during early embryogenesis.5 Zahedi et al. hypothesized LAM as an autoimmune pathology after discovering elevated ANA titers and abnormal serum ribonucleoprotein, immunoglobulin M, and anti-SM antibody in a 28-year-old male with LAM.