It has been found that AXL promotes tumor cell survival by regulating NFκB nuclear translocation, decreasing the activity of pro-apoptotic proteins (BAD and caspase-3) and enhancing the expression of anti-apoptotic markers (survivin, BCL-2 and BCL-XL) (Fig. 1) [70–72]. This evidence concerns the gene NFKB1 and neoplasm.