In contrast, wild-type healthy mouse PEC-B had the lowest expression of Aldh1a2. As shown in Figure 2F, in anti-GBM glomerulonephritis mice, Aldh1a2 expression was progressively and greatly induced in PEC-B, mildly increased in PEC-A2, and was substantially higher on day 5 than on day 1 in PEC-A3 cells, but expression did not change in PEC-A1 cells. Here, ALDH1A2 is linked to glomerulonephritis.