About 85%-90% of those with FH have variants in the LDL receptor (LDLR) gene, however other genes can cause a similar phenotype, including those that encode apolipoprotein B (APOB), proprotein convertase subtilisin–kexin type 9 (PCSK9), and LDL receptor adaptor protein 1 (LDLRAP1) [16–18]. The gene discussed is APOB; the disease is familial hyperaldosteronism.