Therefore, this study aimed to determine if hnRNP A1, hnRNP A2B1, and hnRNP K colocalized with tau aggregates in human brain tissue in a range of tauopathies including AD, mild cognitive impairment (MCI), corticobasal degeneration (CBD), Pick's disease (PiD), and progressive supranuclear palsy (PSP). This evidence concerns the gene HNRNPK and corticobasal degeneration disorder.