Therefore, this study aimed to determine if hnRNP A1, hnRNP A2B1, and hnRNP K colocalized with tau aggregates in human brain tissue in a range of tauopathies including AD, mild cognitive impairment (MCI), corticobasal degeneration (CBD), Pick's disease (PiD), and progressive supranuclear palsy (PSP). This evidence concerns the gene HNRNPK and Classical progressive supranuclear palsy.