This was, in part, based on our previous study showing that other novel pTau interactors identified in our proteomics study strongly colocalize with tau aggregates in AD [33] and further supported by a recent study that showed evidence of increased complexes of hnRNP A2B1‐oligomeric tau in human brain tissue at early stages of AD [5]. This evidence concerns the gene HNRNPA2B1 and Alzheimer disease.