One study showed that hnRNP K did not colocalize with pTau aggregates in the frontal cortex from cases with frontotemporal lobar degeneration with tau pathology (FTLD‐Tau) [11], whereas another showed that hnRNP A2B1 colocalized with oligomeric tau in the temporal cortex of AD patients, primarily at Braak stages II–IV [5]. This evidence concerns the gene MAPT and frontotemporal dementia.