To cite some examples, the induction of macrophage death during the infection sustained by Mycobacterium tuberculosis can promote the spread of the bacterium, while the Fer-1 inhibition of ferroptosis will significantly reduce the bacterial load [32], or the ferroptosis caused by Staphylococcus aureus infection showed that the bacterium causes a lipid membrane damage by interfering with the antioxidant mechanism of GPX4 [25]. Here, GPX4 is linked to infection.