Similar to the single-dose radiotherapy experiment, the combination of VEGF/NRP2 inhibition with conventional fractionation radiotherapy mitigated the growth potential of the tumor (Figure 7A), increased necrosis within the tumor (Figure 7B), and increased the retention of γH2AX (Figure 7C) compared with the IgG with conventional fractionated radiation group. The gene discussed is NRP2; the disease is neoplasm.