HUCMSC-Exos transports miR-24-3p into macrophages, polarizes macrophages to M2 by inhibiting the activation of PLCB3/NF-κB signaling pathway, reduces post-myocardial infarction inflammation, and enhances followed reparative phase, ultimately reducing fibrosis area and improving cardiac function (108). Here, NFKB1 is linked to myocardial infarction.