For instance, following infection by human cytomegalovirus (HCMV), the IFN-β transcript was modified by m6A, and this methylation decreased its half-life, suggesting that HCMV can exploit this control of IFN-β expression to facilitate its replication by increasing m6A modification of the IFN-β transcript and thus decreasing its production (Rubio et al., 2018). Here, IFNB1 is linked to infection.