PRL and pituitary cancer: Interestingly, quantitative reverse transcription polymerase chain reaction (qRT-PCR) results demonstrated that overexpression of exogenous lncRNA UCA1 upregulated HK2 and LDHA mRNA expression, whereas inhibition of lncRNA-UCA1 attenuated the glycolytic pathway, inhibiting prolactin (PRL) production and the proliferation of pituitary cancer cells (46).