FCGR2A and helminthiasis: Identifying the specific immune challenges that shaped FCGR evolution is challenging, but studies on mammalian FCGR3 orthologs indicate that helminth infections exerted significant pressure on the low‐affinity FCGR gene family34 supporting the hypothesis that balancing immunological responses to varying parasitic burdens has been a critical selective pressure at the FCGR loci over evolutionary time.