However, direct evidence on the appearance of AD‐related neuropathological features, like abnormal accumulation of hyperphosphorylated tau protein and amyloid‐beta (Aβ) as well as glia dysfunction and neuroinflammation in the brain (Long & Holtzman, 2019), in human brain post recovery from acute COVID‐19 is still in lack. This evidence concerns the gene MAPT and Alzheimer disease.