Given that ZMYND11 is recognized as an epigenetic reader that specifically recognizes H3.3K36me3 through its PWWP domain,3,4 is dysregulated in cancers including prostate cancer, potentially restricting HNRNPA1-mediated stress granule formation and oncogenic activity, we hypothesized that ZMYND11 might also function as a nonhistone reader, recognizing methylated HNRNPA1 through its MYND reader module. This evidence concerns the gene ZMYND11 and Familial prostate cancer.