Recently, based on experiments in cervical cancer cells (HeLa) and cervical cancer samples, Arellano-Gutiérrez and co-workers have suggested that I3C, through the activation of the AhR, induces UBE2L3 transcription, which might result in the ubiquitination of HPV E7, an element of human papillomavirus and, ultimately, in decreasing cell proliferation [79]. This evidence concerns the gene AHR and cervical cancer.