Interestingly, Staphylococcus epidermidis inhibits C. acnes proliferation by means of the release of succinic acid from glycerol fermentation [56], and also diminishes C. acnes-induced skin inflammation by generating lipoteichoic acid, which suppresses keratinocyte production of Toll-like receptor (TLR) 2, interleukin (IL)-6, and tumor necrosis factor (TNF)-α [57]. The gene discussed is TNF; the disease is dermatitis.