HSPD1 and type 1 diabetes mellitus: Three reasons led to the postulate: (1) shared genetic susceptibilities to both mycobacterial infection and T1D; (2) MAP being the source of the HSP65 protein, providing homology between mycobacterial HSP65 and pancreatic glutamic acid decarboxylase (GAD); and (3) consistent epidemiology findings tying the risk of T1D to early-life exposure to cow’s milk [47,48].