Moreover, DAGs were identified involved in the Golgi apparatus (CNN2, RPGR, GOLGA6L6, GOLGA6L22, GOLGA6L10, GOLGA6L7, GOLGA6L3 pseudogene), Toll-like receptor signaling (PALM3), mRNA splicing (RBM25, CWC25, SF3A1, HELLPAR), apoptosis (MDM4; AP5M1), regulation of NFkB pathway (LIMD1), Wnt–β-catenin–STAT3 signaling (MYH9), aerobic glycolysis (PKM), and methylation (KMT2D, METTL7A, EIF3A, VIRMA), some of which have been associated with progression and prognosis in DLBCL [34,35,36,37,38,39]. The gene discussed is HELLPAR; the disease is diffuse large B-cell lymphoma.