For example, Venetoclax, a B-cell lymphoma (Bcl) homology domain-3 (BH3) mimetic, prevents the complex formation between Bcl2 and proapoptotic BH3-only proteins, thereby destabilizing the outer mitochondrial membrane and inducing apoptosis in cancer cells by mitochondrial swelling, depolarization and fragmentation [62]; findings very similar to those we obtained with AM and DEA. This evidence concerns the gene BCL2 and cancer.