MAG and Ataxia: Afterwards, homozygous and heterozygous variants in MAG were reported in individuals from North African [8], European, and Middle Eastern [10], European [9], and Turkish [11] origins, presenting varying combinations of psychomotor delay, abnormalities in eye movements, oculomotor apraxia, ataxia, spasticity, nystagmus, dystonia, dysfunction of the pyramidal tract, and neuropathic symptoms.