Afterwards, homozygous and heterozygous variants in MAG were reported in individuals from North African [8], European, and Middle Eastern [10], European [9], and Turkish [11] origins, presenting varying combinations of psychomotor delay, abnormalities in eye movements, oculomotor apraxia, ataxia, spasticity, nystagmus, dystonia, dysfunction of the pyramidal tract, and neuropathic symptoms. Here, MAG is linked to cerebellar ataxia.