Human samples of MASLD, MASH, and cirrhosis revealed alterations in splicing patterns, including increased inclusion of the profibrogenic EDA exon (exon 33) in the fibronectin 1 (FN1) gene, exon 23 in the MYO1B gene, as well as exon skipping events in exon 11 of the INSR gene and exon 13 of the SLK gene [82]. This evidence concerns the gene FN1 and Cirrhosis.