To assess the impact of RES treatment on anxiety-like behavior and its metabolic correlates—such as 11β-HSD-1 in the liver, and cerebral MAO-A, MAO-B, and lipid peroxidation (LPO) contents in the brain—as well as on liver injury caused by chronic stress, we initiated a 10-day exposure of rats to predator stress (PS) (Figure 1). This evidence concerns the gene MAOA and Anxiety.