Our preliminary in vitro differential gene expression data revealed an upregulation of genes associated with immune cell chemotaxis including the monocyte chemoattractant CCL2 and the chemokine receptor CXCR3, which has been reported to have a dichotomous role in the tumor microenvironment, but is reported to aid in immune cell infiltration which can lead to tumor suppression [67]. The gene discussed is CCL2; the disease is neoplasm.