Previous research revealed that Slc38a5 was significantly decreased in both Lrp5−/− and Ndpy/− retinas, which are experimental models of familial exudative vitreoretinopathy (FEVR) and Norrie disease, respectively, two genetic vascular eye diseases with inadequate retinal vasculature [182,183,184]. This evidence concerns the gene LRP5 and exudative vitreoretinopathy.