A more comprehensive analysis indicates that upregulated proteins (ELOVL5, ELOVL7, TECR, HSD17B4, ACSL1, HACD2, and CBR4) are significantly enriched within the pathways of oxidative phosphorylation and metabolic pathways pertinent to amyotrophic lateral sclerosis. The gene discussed is TECR; the disease is amyotrophic lateral sclerosis.