The overexpression of FOXM1 in many human cancers is associated with advanced tumor stage, high proliferation rate, aneuploidy, and poor prognosis, as FOXM1 has been experimentally shown to regulate apoptosis, drug resistance, DNA damage repair, stem cell renewal, angiogenesis, metastasis, and mitotic spindle maintenance [108,109,110,111]. This evidence concerns the gene FOXM1 and neoplasm.