In the three types of cancer studied, glioblastoma, lung, and breast cancers, the peptides induced massive apoptotic cell death and induced the overexpression of key proteins in apoptosis, including caspases 3, caspase 8, p53, Cyto c, and SMAC/Diablo, while decreasing the expression of anti-apoptotic proteins, such as Bcl-2 [5,7]. The gene discussed is BCL2; the disease is cancer.