For example, circARHGAP12 was shown to contribute to the development of cervical cancer through the IGF2BP2/FOXM1 pathway, which is dependent on m6A. Moreover, RNA immunoprecipitation (RIP), fluorescence in-situ hybridization (FISH), and Act D experiments confirmed the binding of IGF2BP2 to circARHGAP12 and the ability of IGF2BP2 to increase the stability of circARHGAP12 [26]. Here, FOXM1 is linked to cervical cancer.