MTOR and myocardial infarction: Furthermore, QL has the potential to reinstate cardiac functionality, mitigate cardiac hypertrophy and failure, and enhance mitochondrial function.7, 8, 9 By activating the Peroxisome Proliferator-Activated Receptor-γ (PPAR-γ) and mTOR pathway, respectively, QL can promote cardiac remodeling, thereafter to alleviate Myocardial Infarction (MI) or ischemia-reperfusion injury.10