A lower risk of endometrial-like tissue, as for endometriotic implants, susceptibility to the viral infection has been linked to the lower expression of host proteins related to SARS-CoV-2, such as Transmembrane protease 2 serine protease-2 (TMPRSS2) and especially angiotensin-converting enzyme 2 (ACE2), which is the entry point on the cell [34]. The gene discussed is PRSS2; the disease is viral infectious disease.