Liu et al. [37] found that 24 h in vitro exposure of the human A549 lung adenocarcinoma cell line to hyperoside at concentrations of 20, 40, 60, 80, and 100 μg/mL resulted in concentration-dependent apoptosis via the caspase-3 and P53 signalling pathways. Here, TP53 is linked to lung adenocarcinoma.