Triterpenoids (TER) of Rhus chinensis Mill., with unsignificant cell toxicity (Table 1), dose-dependently inhibited the expression of the glucose metabolism enzymes GLUT1, LDHA, PKM2, and MCT1, which mediate lactate transport, and affect the growth and proliferation of tumor cells by affecting the ASIC2-mediated calcineurin/NFAT1 pathway [73]. The gene discussed is PKM; the disease is neoplasm.