Moreover, by acting as a ceRNA for miR-338-3p, MALAT-1 has been found to directly induce the expression of male-specific lethal 2 homolog (MSL2), which is involved in chromatin organization, maintenance of normal histone profiles and DNA damage response [136], thus indicating a contribution of the MALAT-1/MSL2/miR-338-3p molecular axis in MG. The gene discussed is MSL2; the disease is myasthenia gravis.