TJP1 and congenital rubella syndrome: We found that CXCR2i treatment reversed the brain‐injurious effects of chronic stress as revealed by ameliorated Aβ40 deposition in brain blood vessels (Figure 2H), preserved ZO1 expression (Figure 2I), and attenuated white matter damage (Figure 2J) in both CXCR2i‐treated WT‐ and CAA‐CRS mice (Figure 2H–J).