In this context, AML subtypes that display a transcriptional similarity and co‐cluster with KMT2A‐r, NPM1‐mutant, and NUP98‐r AMLs, including those harboring UBTF‐TD and DEK::NUP214 may also show increased sensitivity towards Menin inhibition.140. Here, KMT2A is linked to acute myeloid leukemia.