Remarkably, replacing the NUP98 N‐terminus by an artificial stretch of 39 FG repeats fused to the C‐terminus of KDM5A (artFG‐KDM5A) preserved its localization to nuclear puncta and maintained its oncogenic function by inducing leukemia‐associated gene expression programs similar to the original NUP98::KDM5A fusion protein.125. This evidence concerns the gene NUP98 and leukemia.