APOB and coronary artery disorder: In our previous study we identified two clusters of SNPs that had differing effects on TRL/remnants versus LDL; the SNP cluster that gave higher genetically predicted TRL/remnant levels was associated with a higher CHD risk per unit increase in apoB.2 The present investigation extended this SNP cluster-based approach to provide a quantitative estimate of the relative atherogenicity of TRL/remnant particles.